/PRNewswire/ -- Colorful pottery may look nice on the dining room table. But beware: it can cause serious harm if it can contaminate food placed in it with lead.
The Food and Drug Administration (FDA) says it has confirmed reports that traditional ceramic pottery made by several manufacturers in Mexico -- and labeled "lead free" -- in fact contains lead.
Learn about this pottery, and steps you can take to detect it, at http://www.fda.gov/ForConsumers/ConsumerUpdates/ucm233531.htm.
-----
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Showing posts with label mexico. Show all posts
Showing posts with label mexico. Show all posts
Friday, November 19, 2010
Thursday, May 21, 2009
H1 N.......what?!!? America's Mexican "Influenza"? asks Researcher Lawrence Broxmeyer, MD
TT Note: Hold on to your flu-like symptoms, folks. This doctor looks at the flu epidemic of 1918 and some researchers' findings. When compared to today's trendy virus, there appears to be some room for debate on the current flu-like symptoms. Is it really the flu?
/24-7 / -- All that is H1N1 does not glitter, at least with respect to the tireless efforts of Virologists, Epidemiologists and Health Officials to stake claim that the current "flu-like illness" pandemic is tied to "Influenza".
"The "H" and "N" of influenza sub-typing" Lawrence Broxmeyer, MD mentions, "revolves around two glycoproteins called Hemagglutin and Neuraminidase, both of which can be, and are, associated with infectious diseases such as the minuscule, viral forms of tuberculosis, a disease which ought to be high on the differential diagnosis for 'flu-like illness' . Since August, 2008, a Medline study in the Journal of Clinical Biochemistry showed that sputum neuraminidase levels over 1.0 mU per mL were proven associated with having tuberculosis in 92% of cases, previous to which bacteria closely related to TB where shown, through crystallization, to produce the same protein neuraminidase used to subtype 'Influenza'."
Furthermore, reminded Lawrence Broxmeyer MD, "Khomenko's 1993 study, showed that the explosive contagiousness of just such influenza-like forms of tuberculosis are exactly the stuff that previous epidemics and pandemics could have been made of." Khomenko was cited by Nobel nominee Lida Mattman in her textbook prior to her untimely death last year.
"That is exactly why", reiterated Lawrence Broxmeyer, MD "that in response to the present world "flu" pandemic, Japan's Health Ministry's Tuberculosis Infection Diseases Control Division deputy director Takeshi Enami went hand in hand with Yoshio Nanba, director of The Office of Pandemic Influenza Preparedness and Response, to attend a news conference in Tokyo on May 1, 2009."
But back in the US, the CDC and NIH seem to feel differently, ignoring everything but "the virus". There was much the same "Influenza" talk when in 1990, a new multi-drug-resistant (MDR) tuberculosis outbreak took place in a large Miami municipal hospital. Soon thereafter, similar outbreaks in three New York City hospitals left many sufferers dying within weeks. By 1992, approximately two years later, drug-resistant tuberculosis had spread to seventeen US states, with mini-epidemics in Florida, Michigan, New York, California, Texas, Massachusetts, and Pennsylvania and was reported, not by the American, but the international media, as out of control. Viral forms of swine, avian and human TB can be transmitted from one species to another. By 1993 the World Health Organization (WHO), proclaimed tuberculosis a global health emergency.
"No one can deny the similarities between the onset of the 1918 epidemic and that of today," mentioned Lawrence Broxmeyer MD, "Yet a Press Release, issued on August 19, 2008, by the National Institute of Allergy and Infectious Diseases (NIAID), contains a striking finding and conclusion: The 20 to 40 million deaths worldwide from the great 1918 Influenza ("Flu") Pandemic were NOT due to "flu" or a virus, but to pneumonia caused by massive bacterial infection."
Subsequently, a study published in JAMA by Talbot and Moore in 2000 showed that Mexican immigrants to the US have the highest case rates for tuberculosis among foreign born persons.
The research of Lawrence Broxmeyer MD first proclaimed that the 1918 pandemic was due to bacteria, particularly mutant forms of flu-like fowl, swine, bovine, and human tuberculosis (TB) bacteria. These forms of tuberculosis are often viral-like, mutate frequently and can "skip" from one species to another. Moreover the antibodies from such viral TB forms react in the compliment fixation and later "viral" assays. They also grow on cultures which are supposed to grow only viruses.
In a supportive 16-page-paper which appeared in Population and Development Review, University of California demographers Andrew Noymer and Michel Garenne came up with convincing statistics showing that undetected tuberculosis may have been the real killer in the 1918 flu epidemic.
Noymer's hypothesis stands sound against history. Few flu "experts" are aware that in medical texts printed circa 1918 "Influenza" was attributed not to a virus but a bacteria called Mycobacterium influenzae, discovered by Pfeiffer and Canon in 1892......not exactly a coincidence since Richard Pfeiffer worked, at one time, for Robert Koch, the discoverer of tuberculosis, a disease also caused by another bacillus called Mycobacterium tuberculosis. Both mycobacteria stained best with carbol-fuchsin, a bacterial stain commonly used in the staining of mycobacteria as it has an affinity for the mycolic acids found in their cell walls. Mycobacteria such as tuberculosis are particularly deadly because they share properties of the fungi ("myco-") as well as bacteria. TB was, not all that long ago, referred to as "captain of the men of death".
Mycobacterium influenzae was considered by most to be the cause of influenza until 1933. But there were serious diagnostic problems with Mycobacterium influenza. Stengel and Fox warned about them in their widely quoted W.B. Saunder's 1915 version of "A Textbook of Pathology". Problems with identification revolved mainly in that although the bacterial influenza occurred abundantly in the sputum of flu patients at first, it decreased in quantity as the cases advanced. And when purulent expectoration stopped Mycobacterium influenzae "disappears entirely".
In 1933 English physicians Wilson Smith, Christopher H. Andrewes, and Patrick P. Laidlaw removed secretions from the throat of a humans with flu-like symptoms thought to have "influenza" , and then filtered out a suspected infectious agent, which by virtue of the fact that it went through a filter was falsely proclaimed to be "a virus". Injecting it into ferrets, the ferrets then developed the same flu-like symptoms which Smith, Andrews and Laidlaw summarily declared as "influenza".
But to government pathologist and pioneer physician/researcher Sir John Crofton (1912-69 ), who by virtue of his office had examined some of Laidlaw's human "Flu" samples, Laidlaw's entire study was flawed. Crofton found Laidlaw's Flu samples to be laced with Mycobacteria influenza, which by then had been renamed Hemophyllis Influenza. Crofton personally confronted and challenged Laidlaw to come to his laboratory for the proof that his Influenza samples weren't viral. However historical and political momentum carried Laidlaw's study through and a great opportunity was missed to correct the record.
In a landmark study, Dr. Robert Donaldson, working out of the Pathological Society of Great Britain had ruled out that the mycobacteria now referred to as H. Influenza by itself was behind 1918, perhaps because of its disappearing nature. Yet at the same time he quickly added that there wasn't "the slightest shred of evidence" that the disease was due to a "virus" or influenza. Nor was Donaldson ever able to refute Broxmeyer and Noymer's feelings that TB was behind the many deaths in the pandemic, specifically because it is well known that secondary bacterial infections, be they from opportunistic Haemophillus influenza or any other common bacteria, are a common secondary manifestation in TB-infected lungs. During the pandemic, one-third of patients who had Haemophillus influenza where also found to have tuberculosis - keeping in mind, as always, that many other cases with TB went undiagnosed.
"In order to understand why we have this emphasis by those virologists invested in a 'killer flu' in the US today", said Lawrence Broxmeyer MD, "one must look back historically at the science itself."
In truth, until the late 1940s influenza 'viruses' were studied as infections, which, although filterable, were conceived of as analogous to bacteria, a kind of ultra or viral-like bacteria.
Not to be deterred, and still seeing Influenza as a great opportunity for virology, in 1941 virologist Hirst claimed that influenza ''virus'' could agglutinate (or clot) red blood cells of fowl and other animal species. The "H" in H1N1 comes to us through Hirst, who showed that ''virus'' particles first adsorbed to the red cells and, after a certain time, eluted again as a result of what could be interpreted as an enzymatic reaction. But 6 years later, Middlebrook and Dubos made this seem nothing more than a cheap hat trick by showing that similarly red blood cell agglutination could be produced by sera from patients with tuberculosis. Takahashi and Ono reviewed similar red cell agglutination occurring in the presence of tuberculous serums.
"As Influenza historian van Helvoort aptly pointed out", summed-up Lawrence Broxmeyer MD, "indeed, in the 1930s and 1940s the concept of 'filterable viruses', including Influenza, were subject to such criticism that Virology's very foundations were threatened. Dogmatized statements like those coming from pioneer virologist Andre Lwoff in 1957 : ''Viruses should be considered as viruses because viruses are viruses'' were totally unacceptable, and did little to help the situation.
So it was in 1952 that Cornelius P. Rhoads, Director of Sloan-Kettering Institute for Cancer Research in New York City, remarked in a conference introduction that the term ''viruses'', such as that of Influenza, had achieved ''a high professional status with doubtful credentials''.
What we have today, mentioned Broxmeyer, is a pandemic with "flu-like" symptoms. And flu-like symptoms dosen't necessarily mean Influenza is the underlying cause.
Reference: Pubmed. Lawrence Broxmeyer MD, Bird flu, influenza and 1918: The case for mutant Avian tuberculosis. Med Hypotheses. 2006;67(5):1006-15. Epub 2006 Jun 27. http://drbroxmeyer.netfirms.com/PDF%20001%20Bird%20Flu%20Editorial.pdf
-----
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/24-7 / -- All that is H1N1 does not glitter, at least with respect to the tireless efforts of Virologists, Epidemiologists and Health Officials to stake claim that the current "flu-like illness" pandemic is tied to "Influenza".
"The "H" and "N" of influenza sub-typing" Lawrence Broxmeyer, MD mentions, "revolves around two glycoproteins called Hemagglutin and Neuraminidase, both of which can be, and are, associated with infectious diseases such as the minuscule, viral forms of tuberculosis, a disease which ought to be high on the differential diagnosis for 'flu-like illness' . Since August, 2008, a Medline study in the Journal of Clinical Biochemistry showed that sputum neuraminidase levels over 1.0 mU per mL were proven associated with having tuberculosis in 92% of cases, previous to which bacteria closely related to TB where shown, through crystallization, to produce the same protein neuraminidase used to subtype 'Influenza'."
Furthermore, reminded Lawrence Broxmeyer MD, "Khomenko's 1993 study, showed that the explosive contagiousness of just such influenza-like forms of tuberculosis are exactly the stuff that previous epidemics and pandemics could have been made of." Khomenko was cited by Nobel nominee Lida Mattman in her textbook prior to her untimely death last year.
"That is exactly why", reiterated Lawrence Broxmeyer, MD "that in response to the present world "flu" pandemic, Japan's Health Ministry's Tuberculosis Infection Diseases Control Division deputy director Takeshi Enami went hand in hand with Yoshio Nanba, director of The Office of Pandemic Influenza Preparedness and Response, to attend a news conference in Tokyo on May 1, 2009."
But back in the US, the CDC and NIH seem to feel differently, ignoring everything but "the virus". There was much the same "Influenza" talk when in 1990, a new multi-drug-resistant (MDR) tuberculosis outbreak took place in a large Miami municipal hospital. Soon thereafter, similar outbreaks in three New York City hospitals left many sufferers dying within weeks. By 1992, approximately two years later, drug-resistant tuberculosis had spread to seventeen US states, with mini-epidemics in Florida, Michigan, New York, California, Texas, Massachusetts, and Pennsylvania and was reported, not by the American, but the international media, as out of control. Viral forms of swine, avian and human TB can be transmitted from one species to another. By 1993 the World Health Organization (WHO), proclaimed tuberculosis a global health emergency.
"No one can deny the similarities between the onset of the 1918 epidemic and that of today," mentioned Lawrence Broxmeyer MD, "Yet a Press Release, issued on August 19, 2008, by the National Institute of Allergy and Infectious Diseases (NIAID), contains a striking finding and conclusion: The 20 to 40 million deaths worldwide from the great 1918 Influenza ("Flu") Pandemic were NOT due to "flu" or a virus, but to pneumonia caused by massive bacterial infection."
Subsequently, a study published in JAMA by Talbot and Moore in 2000 showed that Mexican immigrants to the US have the highest case rates for tuberculosis among foreign born persons.
The research of Lawrence Broxmeyer MD first proclaimed that the 1918 pandemic was due to bacteria, particularly mutant forms of flu-like fowl, swine, bovine, and human tuberculosis (TB) bacteria. These forms of tuberculosis are often viral-like, mutate frequently and can "skip" from one species to another. Moreover the antibodies from such viral TB forms react in the compliment fixation and later "viral" assays. They also grow on cultures which are supposed to grow only viruses.
In a supportive 16-page-paper which appeared in Population and Development Review, University of California demographers Andrew Noymer and Michel Garenne came up with convincing statistics showing that undetected tuberculosis may have been the real killer in the 1918 flu epidemic.
Noymer's hypothesis stands sound against history. Few flu "experts" are aware that in medical texts printed circa 1918 "Influenza" was attributed not to a virus but a bacteria called Mycobacterium influenzae, discovered by Pfeiffer and Canon in 1892......not exactly a coincidence since Richard Pfeiffer worked, at one time, for Robert Koch, the discoverer of tuberculosis, a disease also caused by another bacillus called Mycobacterium tuberculosis. Both mycobacteria stained best with carbol-fuchsin, a bacterial stain commonly used in the staining of mycobacteria as it has an affinity for the mycolic acids found in their cell walls. Mycobacteria such as tuberculosis are particularly deadly because they share properties of the fungi ("myco-") as well as bacteria. TB was, not all that long ago, referred to as "captain of the men of death".
Mycobacterium influenzae was considered by most to be the cause of influenza until 1933. But there were serious diagnostic problems with Mycobacterium influenza. Stengel and Fox warned about them in their widely quoted W.B. Saunder's 1915 version of "A Textbook of Pathology". Problems with identification revolved mainly in that although the bacterial influenza occurred abundantly in the sputum of flu patients at first, it decreased in quantity as the cases advanced. And when purulent expectoration stopped Mycobacterium influenzae "disappears entirely".
In 1933 English physicians Wilson Smith, Christopher H. Andrewes, and Patrick P. Laidlaw removed secretions from the throat of a humans with flu-like symptoms thought to have "influenza" , and then filtered out a suspected infectious agent, which by virtue of the fact that it went through a filter was falsely proclaimed to be "a virus". Injecting it into ferrets, the ferrets then developed the same flu-like symptoms which Smith, Andrews and Laidlaw summarily declared as "influenza".
But to government pathologist and pioneer physician/researcher Sir John Crofton (1912-69 ), who by virtue of his office had examined some of Laidlaw's human "Flu" samples, Laidlaw's entire study was flawed. Crofton found Laidlaw's Flu samples to be laced with Mycobacteria influenza, which by then had been renamed Hemophyllis Influenza. Crofton personally confronted and challenged Laidlaw to come to his laboratory for the proof that his Influenza samples weren't viral. However historical and political momentum carried Laidlaw's study through and a great opportunity was missed to correct the record.
In a landmark study, Dr. Robert Donaldson, working out of the Pathological Society of Great Britain had ruled out that the mycobacteria now referred to as H. Influenza by itself was behind 1918, perhaps because of its disappearing nature. Yet at the same time he quickly added that there wasn't "the slightest shred of evidence" that the disease was due to a "virus" or influenza. Nor was Donaldson ever able to refute Broxmeyer and Noymer's feelings that TB was behind the many deaths in the pandemic, specifically because it is well known that secondary bacterial infections, be they from opportunistic Haemophillus influenza or any other common bacteria, are a common secondary manifestation in TB-infected lungs. During the pandemic, one-third of patients who had Haemophillus influenza where also found to have tuberculosis - keeping in mind, as always, that many other cases with TB went undiagnosed.
"In order to understand why we have this emphasis by those virologists invested in a 'killer flu' in the US today", said Lawrence Broxmeyer MD, "one must look back historically at the science itself."
In truth, until the late 1940s influenza 'viruses' were studied as infections, which, although filterable, were conceived of as analogous to bacteria, a kind of ultra or viral-like bacteria.
Not to be deterred, and still seeing Influenza as a great opportunity for virology, in 1941 virologist Hirst claimed that influenza ''virus'' could agglutinate (or clot) red blood cells of fowl and other animal species. The "H" in H1N1 comes to us through Hirst, who showed that ''virus'' particles first adsorbed to the red cells and, after a certain time, eluted again as a result of what could be interpreted as an enzymatic reaction. But 6 years later, Middlebrook and Dubos made this seem nothing more than a cheap hat trick by showing that similarly red blood cell agglutination could be produced by sera from patients with tuberculosis. Takahashi and Ono reviewed similar red cell agglutination occurring in the presence of tuberculous serums.
"As Influenza historian van Helvoort aptly pointed out", summed-up Lawrence Broxmeyer MD, "indeed, in the 1930s and 1940s the concept of 'filterable viruses', including Influenza, were subject to such criticism that Virology's very foundations were threatened. Dogmatized statements like those coming from pioneer virologist Andre Lwoff in 1957 : ''Viruses should be considered as viruses because viruses are viruses'' were totally unacceptable, and did little to help the situation.
So it was in 1952 that Cornelius P. Rhoads, Director of Sloan-Kettering Institute for Cancer Research in New York City, remarked in a conference introduction that the term ''viruses'', such as that of Influenza, had achieved ''a high professional status with doubtful credentials''.
What we have today, mentioned Broxmeyer, is a pandemic with "flu-like" symptoms. And flu-like symptoms dosen't necessarily mean Influenza is the underlying cause.
Reference: Pubmed. Lawrence Broxmeyer MD, Bird flu, influenza and 1918: The case for mutant Avian tuberculosis. Med Hypotheses. 2006;67(5):1006-15. Epub 2006 Jun 27. http://drbroxmeyer.netfirms.com/PDF%20001%20Bird%20Flu%20Editorial.pdf
-----
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Wednesday, April 29, 2009
The Pig vs. the Census Taker: Who Will Win?
TT Note: Here's an interesting story on the possible source for the Mexican swine flu. Note how the blame for the outbreak is listed as potentially coming from America. It will be interesting to see how this plays out. Of course, if the consensus is a census worker, that will make the current door to door census salesmen types highly suspect. Oops, I forgot, they already are.
SWINE FLU
Mexico tries to focus on source of infection
By Tracy Wilkinson and Cecilia Sánchez
April 28, 2009
Reporting from Mexico City -- With the death toll climbing, Mexican authorities at the center of an international swine flu epidemic struggled Monday to piece together its lethal march, with attention focusing on a 4-year-old boy and a pig farm.
The boy, who survived the illness, has emerged as Mexico's earliest known case of the never-before-seen virus, Health Secretary Jose Angel Cordova said Monday. It provides an important clue to the unique strain's path......http://www.latimes.com/features/health/la-fg-mexico-flu28-2009apr28,0,1701782.story
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SWINE FLU
Mexico tries to focus on source of infection
By Tracy Wilkinson and Cecilia Sánchez
April 28, 2009
Reporting from Mexico City -- With the death toll climbing, Mexican authorities at the center of an international swine flu epidemic struggled Monday to piece together its lethal march, with attention focusing on a 4-year-old boy and a pig farm.
The boy, who survived the illness, has emerged as Mexico's earliest known case of the never-before-seen virus, Health Secretary Jose Angel Cordova said Monday. It provides an important clue to the unique strain's path......http://www.latimes.com/features/health/la-fg-mexico-flu28-2009apr28,0,1701782.story
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Wednesday, July 30, 2008
FDA Extends Consumer Warning on Serrano Peppers from Mexico
Laboratory testing by the U.S. Food and Drug Administration has confirmed that both a sample of serrano pepper and a sample of irrigation water collected by agency investigators on a farm in the state of Nuevo Leon, Mexico, contain Salmonella Saintpaul with the same genetic fingerprint as the strain of bacteria that is causing the current outbreak in the United States.
As a result, until further notice, the FDA is advising consumers to avoid raw serrano peppers from Mexico, in addition to raw jalapeño peppers from Mexico, and any foods that contain them.
The test results announced today are part of the FDA's continuing intensive investigation into the outbreak of Salmonella Saintpaul. The investigation has involved tracing back, through complex distribution channels, the origins of products associated with clusters of illness in the United States, as well as inspections and evaluation of farms and facilities in this country and in Mexico, and the collection and testing of environmental and product samples. One of these tracebacks led to a packing facility in Mexico, and to a particular farm, where the agency obtained the samples.
Previously, FDA inspectors collected a positive sample of jalapeño pepper from a produce-distribution center owned by Agricola Zaragosa in McAllen, Texas. The FDA continues to work on pinpointing where and how in the supply chain this first positive jalapeño pepper sample became contaminated. It originated from a different farm in Mexico than the positive samples of serrano pepper and irrigation water.
The FDA is still analyzing many of the samples taken at various farms in Mexico. If laboratory results warrant, the FDA will provide consumers with additional cautions or warnings necessary to protect their health.
On July 17, the FDA announced it had determined that fresh tomatoes now available in the domestic market are not associated with the current outbreak. As a result, the agency removed its June 7 warning against eating certain types of red raw tomatoes.
Monday, July 28, 2008
U.S. Grown Jalapeño and Serrano Peppers Not Connected to Salmonella Saintpaul Outbreak
The U.S. Food and Drug Administration is advising consumers that jalapeño and Serrano peppers grown in the United States are not connected with the current Salmonella Saintpaul outbreak.
However, the FDA continues to advise consumers to avoid raw jalapeño peppers--and the food that contains them--if they have been grown, harvested or packed in Mexico.
In addition to domestically grown raw jalapeño peppers, commercially canned, pickled and cooked jalapeño peppers from any and all geographic locations also are not connected with the current Salmonella Saintpaul outbreak.
The FDA is working with state regulatory agencies and food industry groups that represent restaurants, grocery stores and wholesalers to ensure everyone clearly understands this new, more narrow, advisory. The FDA will continue to refine its consumer guidance as the agency’s investigation continues.
The more narrow advisory the FDA is issuing today (July 25, 2008) is based on evidence gathered during a multi-week, intensive investigation conducted in partnership with the U.S. Centers for Disease Control and Prevention and public health authorities in several U.S. states to find the source of the contamination that led to the outbreak. The collective review of the current traceback investigation and harvesting dates, matched with the dates that people became ill, have combined to indicate that the contaminated jalapeño pepper originated in Mexico.
Additional traceback and traceforward information obtained last week has led to the determination that the Agricola Zarigoza produce-distribution center in McAllen, Texas--from where FDA took the positive jalapeño pepper sample--was not the original source of the contamination.
The FDA is continuing to advise that people in high-risk populations, such as elderly persons, infants and people with impaired immune systems, avoid eating raw Serrano peppers from Mexico or food made from raw Serrano peppers from Mexico until further notice.
Sunday, December 30, 2007
Violent Border Smugglers Scare U.S. Scientists
Reading a title about violent border smugglers, the first thing I'd think was there were problems in any part of the world but the United States. But no, this article is about our southern border with Mexico. It's hard to imagine trucks full of gun-toting smugglers coming easily across our borders. Reading the article below brought home the seriousness of the problem in ways that none of the other articles I've read have done thus far.
We have a huge problem. Our politicians seem to be making token attempts to placate those who raise a fuss about illegal aliens, drug smuggling and the potential for terrorists to walk across our border. The louder the up swell of our discontent the more token bandaids they plaster. The problem is we don't need bandaids, we need serious surgery along with heavy antibiotics.
I understand it's another one of those huge, complicated problems that could have been solved easily years back. Now it's grown to be such a monster that any solution is going to hurt someone somewhere. Somehow we've got to figure out a way for the business faction, the various political factions and any other factions involved to see the big picture.
Violent Border Smugglers Scare U.S. Scientists
Fox News / PHOENIX — Biologist Karen Krebbs used to study bats in Organ Pipe Cactus National Monument on the Arizona-Mexico border. Then, she got tired of dodging drug smugglers all night.
"I use night-vision goggles, and you could see them very clearly" — caravans of men with guns and huge backpacks full of drugs, trudging through the desert, Krebbs said. After her 10th or 11th time hiding in bushes and behind rocks, she abandoned her research.
http://www.foxnews.com/story/0,2933,319028,00.html
We have a huge problem. Our politicians seem to be making token attempts to placate those who raise a fuss about illegal aliens, drug smuggling and the potential for terrorists to walk across our border. The louder the up swell of our discontent the more token bandaids they plaster. The problem is we don't need bandaids, we need serious surgery along with heavy antibiotics.
I understand it's another one of those huge, complicated problems that could have been solved easily years back. Now it's grown to be such a monster that any solution is going to hurt someone somewhere. Somehow we've got to figure out a way for the business faction, the various political factions and any other factions involved to see the big picture.
Violent Border Smugglers Scare U.S. Scientists
Fox News / PHOENIX — Biologist Karen Krebbs used to study bats in Organ Pipe Cactus National Monument on the Arizona-Mexico border. Then, she got tired of dodging drug smugglers all night.
"I use night-vision goggles, and you could see them very clearly" — caravans of men with guns and huge backpacks full of drugs, trudging through the desert, Krebbs said. After her 10th or 11th time hiding in bushes and behind rocks, she abandoned her research.
http://www.foxnews.com/story/0,2933,319028,00.html
Friday, December 28, 2007
DEWALT Recalls Cordless Drills due to Fire Hazard
The U.S. Consumer Product Safety Commission, in cooperation with the firm named below, today announced a voluntary recall of the following consumer product. Consumers should stop using recalled products immediately unless otherwise instructed.
Name of Product: DEWALT Cordless Drills
Units: About 346,000
Manufacturer: DEWALT Industrial Tool Company, of Towson, Md.
Hazard: The trigger switch of the cordless drill can overheat, posing a fire hazard to consumers.
Incidents/Injuries: DEWALT has received 11 reports of trigger switches overheating. No injuries or property damage have been reported.
Description: This recall involves DEWALT individual cordless drills listed below:
Model Number
Description
Date Codes
DC920 Heavy-Duty XRP(tm) 1/2" (13mm) 18 Volt Cordless Drill/Driver 200723 through 200742
DC930 Heavy-Duty XRP(tm) 1/2" (13mm) 14.4 Volt Cordless Drill/Driver 200625 through 200746
DC935 Heavy-Duty XRP(tm) 1/2" (13mm) 14.4 Volt Cordless Hammerdrill/Drill/Driver 200627 through 200746
DC936 Heavy-Duty XRP(tm) 1/2" (13mm) 14.4 Volt Cordless Hammerdrill/Drill/Driver 200635 through 200746
DC940 Heavy-Duty XRP(tm) 1/2" (13mm) 12 Volt Cordless Drill/Driver 200635 through 200746
The model number is printed on a sticker on the side of the unit. The date code is embossed on the bottom of the unit. Units stamped with an "M" following the date code have been repaired and are not included in this recall. The packaging of repaired drills has a green dot sticker near the UPC label.
Sold at: Wholesale distributors and retailers from about June 2006 through December 2007 for between $180 and $280.
Manufactured in: Mexico
Remedy: Consumers should stop using the drills immediately and contact DEWALT for the location of the nearest service center to receive a free inspection and, if necessary, free repair.
Consumer Contact: Call DEWALT toll-free at (888) 742-9168 between 8 a.m. and 5 p.m. ET Monday through Friday or visit firm's Web site at www.DEWALT.com
Name of Product: DEWALT Cordless Drills
Units: About 346,000
Manufacturer: DEWALT Industrial Tool Company, of Towson, Md.
Hazard: The trigger switch of the cordless drill can overheat, posing a fire hazard to consumers.
Incidents/Injuries: DEWALT has received 11 reports of trigger switches overheating. No injuries or property damage have been reported.
Description: This recall involves DEWALT individual cordless drills listed below:
Model Number
Description
Date Codes
DC920 Heavy-Duty XRP(tm) 1/2" (13mm) 18 Volt Cordless Drill/Driver 200723 through 200742
DC930 Heavy-Duty XRP(tm) 1/2" (13mm) 14.4 Volt Cordless Drill/Driver 200625 through 200746
DC935 Heavy-Duty XRP(tm) 1/2" (13mm) 14.4 Volt Cordless Hammerdrill/Drill/Driver 200627 through 200746
DC936 Heavy-Duty XRP(tm) 1/2" (13mm) 14.4 Volt Cordless Hammerdrill/Drill/Driver 200635 through 200746
DC940 Heavy-Duty XRP(tm) 1/2" (13mm) 12 Volt Cordless Drill/Driver 200635 through 200746
The model number is printed on a sticker on the side of the unit. The date code is embossed on the bottom of the unit. Units stamped with an "M" following the date code have been repaired and are not included in this recall. The packaging of repaired drills has a green dot sticker near the UPC label.
Sold at: Wholesale distributors and retailers from about June 2006 through December 2007 for between $180 and $280.
Manufactured in: Mexico
Remedy: Consumers should stop using the drills immediately and contact DEWALT for the location of the nearest service center to receive a free inspection and, if necessary, free repair.
Consumer Contact: Call DEWALT toll-free at (888) 742-9168 between 8 a.m. and 5 p.m. ET Monday through Friday or visit firm's Web site at www.DEWALT.com
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